A good result but a way to go yet.
Conclusions
These data suggest that BIT225 treatment can counteract Vpu-mediated downregulation of membrane expressed CD28 & CCR7 on CD4+Tcells
BIT225 thus appears to enhance the costimulatory, signal2, of TCR activation by increasing expression of both the receptor, and ligand on CD4+Tcells and MDM, respectively.
These findings, and those of enhanced homing indicate novel attributes of BIT225 Vpu inhibition that suggest enhanced host immune surveillance of HIV
BITT225 treatment may be a valuable addition to future antiretroviral treatment, and inconsideration of eradication strategies.
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