Welcome Jobo. Great to have a real expert here commenting.
Where I think CU6 has a real advantage is their bis-PMSA construct is able to target the tumour better allowing the longer half-life Cu67 to be more effective while minimising off-target side-effects. There have been no serious side-effects seen even using the 12MBq dose - this is 50% above the AC225 and Lu177 dose level that can be tolerated.
I really hope they push the Cu67 dose to MTD, but it looks like they aren't planning this. It is much easier to convince a clinician to switch to a treatment with superior efficacy than it is to convince them to switch to a new treatment with equal efficacy and less side effects.
CU6 - General discussion, page-292
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