Ben,
read the abstract of the link you provided (the article clearly acknowledges that NAMDR antagonists have the psychosis side effect)
it seems to indicate that focusing on glutamate binding sites as opposed to aspartate receptors is preferential to address the psychotic effects of NAMDR antagonists (aspartate receptor antagonists)
from the announcement above AGN indicate a unique MOA for AGN-007 from other competitors. Furthermore, on the company website the MOA described indicates its effect on glutamate receptors and numerous other areas. it also makes no mention of action on NAMDR
not calling you out here just genuinely interested as i have been bitten before by a band of loyal followers shouting down a naysayer
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