And yet it seems we put so much value in previous results from mouse models. Very strange how these things work. I would love to understand the actual mechanism of how the fpp is injected and then finds its way to the target cell and enters. I guess the same mechanism is at play in the eye. In that case how are we penetrating all the cells we need to penetrate. I assume the fpp is being absorbed by any cell it touches, hence the benefit of limited cell exposure in the eye. I assume to kill a cancer tumour fpp and imyc would need to enter every cell within the tumour, or is there some sort of propagation of effect if that is the correct term. Just general musings, not necessarily asking for anyones best guess.
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