Below is a preprint (needs to be peer reviewed).
A computer simulation
Artemisinin andDerivatives Portray More Potent Binding to Lys353 and Lys31-Binding Hotspots ofSARS-CoV-2 Spike Protein than Hydroxychloroquine.
Interestingly they predict that Artensunate is unlikely to act as a good inhibitor, in its current form, to SARS-Co-2 S Protein. IMO This may support ArTiMist which delivers Artemether and Artenimol after FP metabolism. They suggest more focus on Artenimol because it has good clinical records and most Artemisinin derivatives are converted to this compound once incorporated in the body.
https://chemrxiv.org/articles/In-silico_Studies_of_Antimalarial-agent_Artemisinin_and_Derivatives_Portray_More_Potent_Binding_to_Lys353_and_Lys31-Binding_Hotspots_of_SARS-CoV-2_Spike_Protein_than_Hydroxychloroquine_Potential_Repurposing_of_Artenimol_for_COVID-19_/12098652
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