Just on mouse models & MS it is interesting but the real outcome would be seen when it moves to humans. You have to remember the story of Glatiramer over many years and how incredible outcomes in mice could not be replicated in humans although it could still provide a benefit in copaxone. In the early 70's Israeli researchers were trying to make artificial substances similar to myelin base protein to induce experimental autoimmune encephalomyelitis in mice - Glatiramer was one of these with its 4 amino acids and while it was found to not cause EAE's in the mice it stopped them from getting the disease & cured those that already had it.
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