FTO ameliorates doxorubicin-induced cardiotoxicity by inhibiting ferroptosis via P53–P21/Nrf2 activation in a HuR-dependent m6A manner
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10862061/
if FTO expression reduces Dox induced cardiotoxicity then the inverse is likely true for an FTO inhibitor.
So Bisantrene self rescues cardiotoxicity, anthracycline induced cardiotoxicity and FTO upregulated cardiotoxicity.
FTO ameliorates doxorubicin-induced cardiotoxicity by inhibiting...
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