AutoimmuneOncogenesis and autoimmunity as a result of mRNA...

Autoimmune

• Oncogenesis and autoimmunity as a result of mRNA COVID-19 vaccination
  https://www.authorea.com/users/455597/articles/737938-oncogenesis-and-autoimmunity-as-a-result-of-mrna-covid-19-vaccination
  (“In this paper, we have provided an extensive review of the role of Treg cells in the immune system, with a particular focus on the apparent disruption of their behavior caused by the mRNA vaccines. It appears that the vaccines typically induce an intense IgG antibody response due to the toxicity of the spike protein, along with an extreme inflammatory response through cytokine release by T cells, and, ultimately, the potential for autoantibodies to attack the tissues through recognition of non-self spike protein on the cell surface.” Explanatory article here.)

• The Potential Role of SARS-CoV-2 Infection and Vaccines in Multiple Sclerosis Onset and Reactivation: A Case Series and Literature Review
  https://www.mdpi.com/1999-4915/15/7/1569
  (Authors reported numerous cases of either flares of existing MS or de novo disease in patients who either had SARS-CoV-2 infection, vaccination, and likely both. It is well known that other viral infections can flare MS, however, there were clear-cut cases of mRNA vaccines causing new cases of MS. Paper suggests some de novo cases were completely avoidable by declining COVID-19 vaccination from the start. Explanatory article here.)

• Safety of vaccination against SARS-CoV-2 in people with rheumatic and musculoskeletal diseases: results from the EULAR Coronavirus Vaccine (COVAX) physician-reported registry
  https://ard.bmj.com/content/81/5/695
  (A rheumatologic database published in the BMJ showed that 37% of patients had an adverse response to COVID vaccination, and 4.4% of those vaccinated experienced an exacerbation of a pre-existing autoimmune condition. Explanatory article here.)

• Autoimmune inflammatory reactions triggered by the COVID-19 genetic vaccines in terminally differentiated tissues
  https://www.tandfonline.com/doi/full/10.1080/08916934.2023.2259123
  (The conclusions are inescapable, mRNA vaccines will cause autoimmunity in all applications. "Numerous studies report the onset of autoimmune reactions following COVID-19 vaccination. The histopathological data provide indisputable evidence that demonstrates that the genetic vaccines exhibit an off-target distribution, causing the synthesis of the spike protein and thus triggering autoimmune inflammatory reactions” Explanatory article here.)

• Role of the antigen presentation process in the immunization mechanism of the genetic vaccines against COVID-19 and the need for biodistribution evaluations
  https://pubmed.ncbi.nlm.nih.gov/35298029/
  (All cells that take up mRNA express foreign proteins on the cell surface inviting an immediate auto-immune attack on cells harboring the mRNA and it’s protein products.)

• New-onset autoimmune phenomena post-COVID-19 vaccination
  https://onlinelibrary.wiley.com/doi/10.1111/imm.13443
  (Conclusion: emerging evidence has indicated that new onset of autoimmune manifestations including VITT, autoimmune liver diseases, GBS and IgA nephropathy appears to be associated with COVID-19 vaccines. The plausible mechanisms include molecular mimicry, the production of particular autoantibodies and the role of certain vaccine adjuvants. Further studies are warranted.)

• IgA Vasculitis Following COVID-19 Vaccination: A French Multicenter Case Series Including 12 Patients
  https://www.jrheum.org/content/50/2/252.long
  (The major points of this paper are: 1) auto-immune disease will happen after genetic vaccinations of any type and IgA vasculitis is just the tip of the iceberg, 2) skin rashes can be the only clue to internal organ damage and the need for treatment. Explanatory article here.)

• Autoimmune inflammatory reactions triggered by the COVID-19 genetic vaccines in terminally differentiated tissues
  https://www.tandfonline.com/doi/full/10.1080/08916934.2023.2259123
  (Production of a foreign protein in the human body has turned out to be a disaster as illustrated in paper. Reasons why: 1) each cell that takes up the vaccine expresses the protein in the cell surface initiating autoimmune attack, 2) the tissue distribution appears to be wide involving organs where this attack could be lethal (heart, brain, bone marrow, etc.), 3) both the genetic material and the Spike protein are long lasting (months to years) which is long enough to cause an autoimmune syndrome which may be permanent. Explanatory article here.)

• Molecular mimicry between SARS-CoV-2 spike glycoprotein and mammalian proteomes: implications for the vaccinehttps://link.springer.com/article/10.1007/s12026-020-09152-6
  ("Finally, this study once more reiterates the concept that only vaccines based on minimal immune determinants, unique to pathogens and absent in the human proteome, might offer the possibility of safe and efficacious vaccines." In other words, vaccines need to eliminate the regions of the Spike protein that mimic human proteins in order to avoid triggering autoimmunity.)

• Incidence of Guillain-Barré Syndrome After COVID-19 Vaccination in the Vaccine Safety Datalink
  https://pubmed.ncbi.nlm.nih.gov/35471572/(explanatory article: The incidence rate of confirmed cases per 100,000 person-years was 34.6 during the 1 to 21 days after administration, much higher than the historical background rate of 2 per 100,000 person-years.)

• Pathogenic priming likely contributes to serious and critical illness and mortality in COVID-19 via autoimmunity
  https://pubmed.ncbi.nlm.nih.gov/32292901/
  (The COVID-19 vaccination program causes Disease Enhancement, likely via numerous possible means: from molecular mimicry leading to autoimmunity, or antibody-dependent enhancement. Explanatory article)

• Pathogenic antibodies induced by spike proteins of COVID-19 and SARS-CoV viruseshttps://europepmc.org/article/PPR/PPR357777
  (Preprint article found that “these pathogenic antibodies, through a mechanism of Antibody Dependent Auto-Attack (ADAA), target and bind to host vulnerable cells or tissues such as damaged lung epithelium cells, initiate a self-attack immune response, and lead to serious conditions including ARDS, cytokine release, and death. Moreover, the pathogenic antibodies also induced inflammation and hemorrhage of the kidneys, brain, and heart. Furthermore, the pathogenic antibodies can bind to un-matured fetal tissues and cause abortions, postpartum labors, still births, and neonatal deaths of pregnant mice.”)