Thanks for the interesting info there MY. Quick question re this type of stem cell transplants if I may. I can see how an introduced stem cell which itself isn't at risk would propagate itself and thus its "offspring" (for lack of a better word at this time of the morning) would also not be at risk of infection, but what fails to compute in my little brain is how this protects cells redived from existing stems cells within the body. Presumably the host body still has shoot loads of other cells which remain at risk due to being derived from native stem cells which are inherrantly vunerable.
In my relatively simple logic, introducing new cells which aren't at risk doesn't remove the native cells which are at risk and thus harbour the virus and allow it to propagate. Is it simply that the introduced cells can continue to feed the imune system and thus fight the virus and keep the viral load level low? If this is the case then one assumes that the cycle can still only be broken by allowing the body to kill off the virus in the reseviors which is of course where BIT225 comes in.
Sorry if this is a silly question.
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