But than if inflammation is preventing iron from leaving the cell, and therefore accumulating within the cell, causing the cell to die, what is the cause of the inflammation ?.
In the recent poster of the new data, it showed ATH had a much better affinity to ferrous iron compared to other drugs, the iron that causes all the issues, and also showed much better cellular movement due to its small molecular size.
Therefore, all the research at this point in time shows ATH434 is a better iron chelator with much less side effects, well with only known 1 side effect to date being headaches.
Overall, can ATH434 remove excess iron from inflammed cells before cellular death...it showed it can in the primate study.
In addition to this ATH434 has also been shown to protect mitochondria cellular walls...once again, everything is so positive around ATH434.
within 7 weeks, or 35 business days we will have a glimpse of how the human studies are going.
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