I want just underline that ( IMO) ROS caused mitochondrial damage but PBT2 permanently blocked ROS production and it's toxic effects. Very likely the mechanism in brain is the same and explains why the mitochondria do not work in AD. So we have genetic mitochondria problems but most of likely the main reason is not genetic but related to ROS production which can be controlled by metal balance or treated by correcting metal balance.
This paper was sent for publication on 2nd of Aug. So Prana has known these results already in the summer. It is IMO very natural that now the Italian group and Prana would be planning a clinical study in this diagnosis. Could be an orphan disease. Could be one of the " more acute diagnoses".
So ROS destroys mitochondria !
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